[1] |
Nahrendorf M,Swirski F,Aikawa E,et al.The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions[J].J Exp Med,2007,204(12):3037-3047.
|
[2] |
van Furth R,Cohn ZJ,Joem T.The origin and kinetics of mononuclear phagocytes[J].J Exp Med,1968,128(3):415-435.
|
[3] |
Samokhvalov IM.Deconvoluting the ontogeny of hematopoietic stem cells[J].Cell Mol Life Sci,2014,71(6):957-978.
|
[4] |
Schulz C,Gomez Perdiguero E,Chorro L,et al.A lineage of myeloid cells independent of Myb and hematopoietic stem cells[J].Science,2012,336(6077):86-90.
|
[5] |
Gordon S,Martinez FO.Alternative activation of macrophages:mechanism and functions[J].Immunity,2010,32(5):593-604.
|
[6] |
Mantovani A,Sica A,Sozzani S,et al.The chemokine system in diverse forms of macrophage activation and polarization[J].Trends Immunol,2004,25(12):677-686.
|
[7] |
Nahrendorf M,Swirski FK.Abandoning M1/M2 for a Network Model of Macrophage Function[J].Circ Res,2016,119(3):414-417.
|
[8] |
Walter W,Alonso-Herranz L,Trappetti V,et al.Deciphering the Dynamic Transcriptional and Post-transcriptional Networks of Macrophages in the Healthy Heart and after Myocardial Injury[J].Cell Rep,2018,23(2):622-636.
|
[9] |
Varga T,Mounier R,Horvath A,et al.Highly Dynamic Transcriptional Signature of Distinct Macrophage Subsets during Sterile Inflammation,Resolution,and Tissue Repair[J].J Immunol,2016,196(11):4771-4782.
|
[10] |
Schlundt C,El Khassawna T,Serra A,et al.Macrophages in bone fracture healing:Their essential role in endochondral ossification[J].Bone,2018,106:78-89.
|
[11] |
Lavin Y,Winter D,Blecher-Gonen R,et al.Tissue-resident macrophage enhancer landscapes are shaped by the local microenvironment[J].Cell,2014,159(6):1312-1326.
|
[12] |
Epelman S,Lavine K,Beaudin A,et al.Embryonic and adult-derived resident cardiac macrophages are maintained through distinct mechanisms at steady state and during inflammation[J].Immunity,2014,40(1):91-104.
|
[15] |
Epelman S,Lavine K,Randolph GJ.Origin and functions of tissue macrophages[J].Immunity,2014,41(1):21-35.
|
[32] |
Chen M,Li X,Wang S,et al.The Role of Cardiac Macrophage and Cytokines on Ventricular Arrhythmias[J].Front Physiol,2020,11:1113.
|
[13] |
Lavine K,Epelman S,Uchida K,et al.Distinct macrophage lineages contribute to disparate patterns of cardiac recovery and remodeling in the neonatal and adult heart[J].Proc Natl Acad Sci U S A,2014,111(45):16029-16034.
|
[14] |
Leid J,Carrelha J,Boukarabila H,et al.Primitive Embryonic Macrophages are Required for Coronary Development and Maturation[J].Circ Res,2016,118(10):1498-1511.
|
[16] |
Bajpai G,Bredemeyer A,Li W,et al.Tissue Resident CCR2-and CCR2+Cardiac Macrophages Differentially Orchestrate Monocyte Recruitment and Fate Specification Following Myocardial Injury[J].Circ Res,2019,124(2):263-278.
|
[17] |
Hulsmans M,Clauss S,Xiao L,et al.Macrophages Facilitate Electrical Conduction in the Heart[J].Cell,2017,169(3):510-522,e520.
|
[18] |
Fairweather D,Rose NR.Coxsackievirus-induced myocarditis in mice:a model of autoimmune disease for studying immunotoxicity[J].Methods,2007,41(1):118-122.
|
[19] |
Bao J,Sun T,Yue Y,et al.Macrophage NLRP3 inflammasome activated by CVB3 capsid proteins contributes to the development of viral myocarditis[J].Mol Immunol,2019,114:41-48.
|
[20] |
Li K,Xu W,Guo Q,et al.Differential macrophage polarization in male and female BALB/c mice infected with coxsackievirus B3 defines susceptibility to viral myocarditis[J].Circ Res,2009,105(4):353-364.
|
[21] |
Cihakova D,Barin J,Afanasyeva M,et al.Interleukin-13 protects against experimental autoimmune myocarditis by regulating macrophage differentiation[J].Am J Pathol,2008,172(5):1195-1208.
|
[22] |
Shen Y,Zhang F,Wei XJM,et al.Truncated monocyte chemoattractant protein-1 can alleviate cardiac injury in mice with viral myocarditis via infiltration of mononuclear cells[J].Microbiol Immunol,2014,58(3):195-201.
|
[23] |
Peet C,Ivetic A,Bromage D,et al.Cardiac monocytes and macrophages after myocardial infarction[J].Cardiovasc Res,2020,116(6):1101-1112.
|
[24] |
Mia M,Cibi D,Abdul Ghani S,et al.YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction[J].PLoS Biol,2020,18(12):e3000941.
|
[25] |
Liu S,Chen J,Shi J,et al.M1-like macrophage-derived exosomes suppress angiogenesis and exacerbate cardiac dysfunction in a myocardial infarction microenvironment[J].Basic Res Cardiol,2020,115(2):22.
|
[26] |
Jung M,Ma Y,Iyer R,et al.IL-10 improves cardiac remodeling after myocardial infarction by stimulating M2 macrophage polarization and fibroblast activation[J].Basic Res Cardiol,2017,112(3):33.
|
[27] |
Jia D,Jiang H,Weng X,et al.Interleukin -35 Promotes Macrophage Survival and Improves Wound Healing After Myocardial Infarction in Mice[J].Circ Res,2019,124(9):1323-1336.
|
[28] |
Dick S,Macklin J,Nejat S,et al.Self-renewing resident cardiac macrophages limit adverse remodeling following myocardial infarction[J].Nat Immunol,2019,20(1):29-39.
|
[29] |
Wang J,Seo M,Deci M,et al.Effect of CCR2 inhibitorloaded lipid micelles on inflammatory cell migration and cardiac function after myocardial infarction[J].Int J Nanomedicine,2018,13:6441-6451.
|
[30] |
Lavine K,Pinto A,Epelman S,et al.The Macrophage in Cardiac Homeostasis and Disease:JACC Macrophage in CVD Series (Part 4)[J].J Am Coll Cardiol,2018,72(18):2213-2230.
|
[31] |
Sugita J,Fujiu K,Nakayama Y,et al.Cardiac macrophages prevent sudden death during heart stress[J].Nat Commun,2021,12(1):1910.
|